Volume 6, Issue 4 (Spring 2017)                   J. Aqua. Eco 2017, 6(4): 1-9 | Back to browse issues page

XML Persian Abstract Print


Download citation:
BibTeX | RIS | EndNote | Medlars | ProCite | Reference Manager | RefWorks
Send citation to:

Pakravan S, Akbarzadeh A. The review of pathogenic mechanism of Aeromonas hydrophila and action of tetracycline against it in aquatic animals. J. Aqua. Eco 2017; 6 (4) :1-9
URL: http://jae.hormozgan.ac.ir/article-1-599-en.html
Abstract:   (5964 Views)
Aeromonas hydrophila is one of common bacterial disease in aquatic animals and its outbreak cause to decrease of aquatic production. Aeromonas disease is due to a protein toxin, aerolysin that exported by Aeromonas hydrophila. This protein toxin forms channels on target cells membrane, disrupting normal activities and cause to destruction and death of them. Aerolysin toxic protein is secreted by expression of the aerolysin gene. Aerolysin gene is 1482 bp and it’s an important and stable molecular marker for detecting possible of Aeromonas hydrophila infection. The PCR technique was used to study of this molecular marker. Different antibiotics used to control and treat of Aeromonas disease and tetracycline is one of the importances of them. Antibacterial mechanism of tetracycline is interference in the translation of aerolysin gene and therefore inhibition of protein synthesis in ribosome of bacteria, therefore they condemn dying. Knowledge of the antimicrobial mechanism of antibiotics can play an important role in finding suitable alternative materials for these synthetic drugs.
 
Full-Text [PDF 534 kb]   (2934 Downloads)    
Type of Study: Research | Subject: Special
Published: 2017/06/30

Add your comments about this article : Your username or Email:
CAPTCHA

Rights and permissions
Creative Commons License This work is licensed under a Creative Commons Attribution-NonCommercial 4.0 International License.

© 2024 All Rights Reserved | Journal of Aquatic Ecology

Designed & Developed by : Yektaweb

Creative Commons License
This work is licensed under a Creative Commons — Attribution-NonCommercial 4.0 International (CC BY-NC 4.0)